COVID-19 may cause diabetes by inducing fat cells to malfunction.

Is the Corona virus capable of causing diabetes?
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Fat cells may malfunction as a result of the coronavirus.


COVID-19 has been linked to an increased risk of diabetes in people, according to Science News.


Several studies have suggested that COVID-19 infections may cause diabetes in some people, but exactly why has remained a mystery, according to Live Science. COVID-19 has been related to type 1 and type 2 diabetes, in which the body assaults the pancreas' insulin-producing cells, and type 2 diabetes, in which the pancreas still makes insulin. Still, the cells don't respond appropriately to it. Insulin decreases blood glucose levels by instructing cells to take up and utilize sugar as a fuel source.


The virus that causes COVID-19, SARS-CoV-2, may infiltrate insulin-producing cells in the pancreas, according to some data. That would suggest that the virus damages these cells and reduces insulin secretion, causing some people's blood sugar levels to rise. However, a newer study reveals that these pancreatic cells still function normally in many COVID-19 individuals with extremely high blood sugar levels, suggesting that there must be another explanation, according to Science News.


According to new research conducted by Weill Cornell Medicine physician-scientist Dr. James Lo, blood sugar surges may be linked to low levels of the hormone adiponectin. Adiponectin increases the sensitivity of cells to insulin so that the two act together to control blood sugar levels. Researchers showed that in patients with severe COVID-19 and high blood sugar, fat cells malfunction and release far less adiponectin than normal.


The researchers found a link between severe coronavirus infections and a condition known as acute respiratory distress syndrome, or ARDS, in which fluid accumulates in the lungs' air sacs. The researchers compared this group of COVID-19 patients to those who had experienced ARDS due to other illnesses. Super high blood sugar was found in both groups. Still, only COVID-19 patients had low adiponectin levels, which suggests that the coronavirus somehow altered the hormone, according to researchers who just revealed their findings.


The researchers discovered that it wasn't just adiponectin that went haywire in these COVID-19 individuals. This shows that the fat cells' function had been disrupted.


The researchers concluded that adiponectin might no longer be able to sensitize COVID-19 patients' cells to insulin due to this dysfunction.


Live Science previously revealed that studies have shown that SARS-CoV-2 can directly infect fat cells, and Lo's team confirmed this conclusion in experiments with hamsters and cells in laboratory dishes. This viral invasion of fat cells could disrupt hormone production. According to Science News, infections can cause inflammation, which may impair fat cells' ability to create adiponectin.


SARS-CoV-2 destroys fat cells and modifies their function, and these functional alterations may contribute to the establishment of diabetes in some patients, according to the team's findings. However, this study cannot verify that a drop in fat-derived hormones triggers COVID-19-related diabetes; further research is needed to confirm this underlying process.


Discover the findings in Science News.





Reference : https://www.livescience.com/covid19-may-drive-diabetes-by-damaging-fat-cells

Image source : https://pixabay.com/id/vectors/corona-tanda-simbol-prohibitory-5231110/

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